Detalhe da pesquisa
1.
Long-term effects of amyloid-beta deposits in human iPSC-derived astrocytes.
Mol Cell Neurosci
; 125: 103839, 2023 06.
Artigo
Inglês
| MEDLINE | ID: mdl-36907531
2.
Intracellular deposits of amyloid-beta influence the ability of human iPSC-derived astrocytes to support neuronal function.
J Neuroinflammation
; 20(1): 3, 2023 Jan 03.
Artigo
Inglês
| MEDLINE | ID: mdl-36593462
3.
Amyloid-ß accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism.
J Neuroinflammation
; 20(1): 43, 2023 Feb 20.
Artigo
Inglês
| MEDLINE | ID: mdl-36803838
4.
Altered Distribution of SNARE Proteins in Primary Neurons Exposed to Different Alpha-Synuclein Proteoforms.
Cell Mol Neurobiol
; 43(6): 3023-3035, 2023 Aug.
Artigo
Inglês
| MEDLINE | ID: mdl-37130995
5.
Crosstalk between astrocytes and microglia results in increased degradation of α-synuclein and amyloid-ß aggregates.
J Neuroinflammation
; 18(1): 124, 2021 Jun 03.
Artigo
Inglês
| MEDLINE | ID: mdl-34082772
6.
Astrocytes have the capacity to act as antigen-presenting cells in the Parkinson's disease brain.
J Neuroinflammation
; 17(1): 119, 2020 Apr 16.
Artigo
Inglês
| MEDLINE | ID: mdl-32299492
7.
Human Astrocytes Transfer Aggregated Alpha-Synuclein via Tunneling Nanotubes.
J Neurosci
; 37(49): 11835-11853, 2017 12 06.
Artigo
Inglês
| MEDLINE | ID: mdl-29089438
8.
The Aß protofibril selective antibody mAb158 prevents accumulation of Aß in astrocytes and rescues neurons from Aß-induced cell death.
J Neuroinflammation
; 15(1): 98, 2018 Mar 28.
Artigo
Inglês
| MEDLINE | ID: mdl-29592816
9.
Secretion and Uptake of α-Synuclein Via Extracellular Vesicles in Cultured Cells.
Cell Mol Neurobiol
; 38(8): 1539-1550, 2018 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-30288631
10.
Extensive uptake of α-synuclein oligomers in astrocytes results in sustained intracellular deposits and mitochondrial damage.
Mol Cell Neurosci
; 82: 143-156, 2017 07.
Artigo
Inglês
| MEDLINE | ID: mdl-28450268
11.
Alpha-synuclein oligomer-selective antibodies reduce intracellular accumulation and mitochondrial impairment in alpha-synuclein exposed astrocytes.
J Neuroinflammation
; 14(1): 241, 2017 Dec 11.
Artigo
Inglês
| MEDLINE | ID: mdl-29228971
12.
Slow degradation in phagocytic astrocytes can be enhanced by lysosomal acidification.
Glia
; 63(11): 1997-2009, 2015 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-26095880
13.
The novel steroidal alkaloids dendrogenin A and B promote proliferation of adult neural stem cells.
Biochem Biophys Res Commun
; 446(3): 681-6, 2014 Apr 11.
Artigo
Inglês
| MEDLINE | ID: mdl-24406163
14.
An emerging phenotype of Xq22 microdeletions in females with severe intellectual disability, hypotonia and behavioral abnormalities.
J Hum Genet
; 59(6): 300-6, 2014 Jun.
Artigo
Inglês
| MEDLINE | ID: mdl-24646727
15.
A case report of two male siblings with autism and duplication of Xq13-q21, a region including three genes predisposing for autism.
Eur Child Adolesc Psychiatry
; 23(5): 329-36, 2014 May.
Artigo
Inglês
| MEDLINE | ID: mdl-23974867
16.
Astrocytic accumulation of tau fibrils isolated from Alzheimer's disease brains induces inflammation, cell-to-cell propagation and neuronal impairment.
Acta Neuropathol Commun
; 12(1): 34, 2024 Feb 26.
Artigo
Inglês
| MEDLINE | ID: mdl-38409026
17.
Altered amyloid-ß structure markedly reduces gliosis in the brain of mice harboring the Uppsala APP deletion.
Acta Neuropathol Commun
; 12(1): 22, 2024 Feb 05.
Artigo
Inglês
| MEDLINE | ID: mdl-38317196
18.
Astrocytic uptake of neuronal corpses promotes cell-to-cell spreading of tau pathology.
Acta Neuropathol Commun
; 11(1): 97, 2023 06 17.
Artigo
Inglês
| MEDLINE | ID: mdl-37330529
19.
Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice.
J Neuroinflammation
; 9: 17, 2012 Jan 23.
Artigo
Inglês
| MEDLINE | ID: mdl-22269349
20.
CRISPR-Cas9 treatment partially restores amyloid-ß 42/40 in human fibroblasts with the Alzheimer's disease PSEN 1 M146L mutation.
Mol Ther Nucleic Acids
; 28: 450-461, 2022 Jun 14.
Artigo
Inglês
| MEDLINE | ID: mdl-35505961